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arXiv:2505.14120

Disentangling signalling and causal influence

Kathleen Barsse, Paolo Perinotti, Alessandro Tosini, Leonardo Vaglini

Published May 20, 2025• 0 citations
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Abstract

The causal effects activated by a quantum interaction are studied, modelling the last one as a bipartite unitary channel. The two parties, say Alice and Bob, can use the channel to exchange messages -- i.e. to signal. On the other hand, the most general form of causal influence includes also the possibility for Alice, via a local operation on her system, to modify Bob's correlations and viceversa. The presence or absence of these two effects are equivalent, but when they both occur, they can differ in their magnitude. We study the properties of two functions that quantify the amount of signalling and causal influence conveyed by an arbitrary unitary channel. The functions are proved to be continuous and monotonically increasing with respect to the tensor product of channels. Monotonicity is instead disproved in the case of sequential composition. Signalling and causal influence are analytically computed for the quantum SWAP and CNOT gates, in the single use scenario, in the $n$-parallel uses scenario, and in the asymptotic regime. A finite gap is found between signalling and causal influence for the quantum CNOT, thus proving the existence of extra causal effects that cannot be explained in terms of communication only. However, the gap disappears in the asymptotic limit of an infinite number of parallel uses, leaving room for asymptotic equivalence between signalling and causal influence.

DOI
10.48550/arxiv.2505.14120

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The causal effects activated by a quantum interaction are studied, modelling the last one as a bipartite unitary channel. The two parties, say Alice and Bob, can use the channel to exchange messages -- i.e. to signal. On the other hand, the most general form of causal influence includes also the possibility for Alice, via a local operation on her system, to modify Bob's correlations and viceversa. The presence or absence of these two effects are equivalent, but when they both occur, they can differ in their magnitude. We study the properties of two functions that quantify the amount of signalling and causal influence conveyed by an arbitrary unitary channel. The functions are proved to be continuous and monotonically increasing with respect to the tensor product of channels. Monotonicity is instead disproved in the case of sequential composition. Signalling and causal influence are analytically computed for the quantum SWAP and CNOT gates, in the single use scenario, in the nnn-parallel uses scenario, and in the asymptotic regime. A finite gap is found between signalling and causal influence for the quantum CNOT, thus proving the existence of extra causal effects that cannot be explained in terms of communication only. However, the gap disappears in the asymptotic limit of an infinite number of parallel uses, leaving room for asymptotic equivalence between signalling and causal influence.
A causal relation between quantum agents, say Alice and Bob, is necessarily mediated by an interaction. Modeling the latter as a reversible quantum channel, an intervention of Alice can have causal influence on Bob's system, modifying correlations between Alice and Bob's systems. Causal influence between quantum systems necessarily allows for signaling. Here we prove a mismatch between causal influence and signaling via direct computation of the two quantities for the Cnot gate. Finally we show a continuity theorem for causal effects of unitary channels: a channel has small causal influence if it allows for small signaling. Published by the American Physical Society 2024
We study the relation of causal influence between input systems of a reversible evolution and its output systems, in the context of operational probabilistic theories. We analyse two different definitions that are borrowed from the literature on quantum theory—where they are equivalent. One is the notion based on signalling, and the other one is the notion used to define the neighbourhood of a cell in a quantum cellular automaton. The latter definition, that we adopt in the general scenario, turns out to be strictly weaker than the former: it is possible for a system to have causal influence on another one without signalling to it. Remarkably, the counterexample comes from classical theory, where the proposed notion of causal influence determines a redefinition of the neighbourhood of a cell in cellular automata. We stress that, according to our definition, it is impossible anyway to have causal influence in the absence of an interaction, e.g. in a Bell-like scenario. We study various conditions for causal influence, and introduce the feature that we call no interaction without disturbance , under which we prove that signalling and causal influence coincide. The proposed definition has interesting consequences on the analysis of causal networks, and leads to a revision of the notion of neighbourhood for classical cellular automata, clarifying a puzzle regarding their quantisation that apparently makes the neighbourhood larger than the original one.
We examine constraints on quantum operations imposed by relativistic causality. A bipartite superoperator is said to be localizable if it can be implemented by two parties (Alice and Bob) who share entanglement but do not communicate; it is causal if the superoperator does not convey information from Alice to Bob or from Bob to Alice. We characterize the general structure of causal complete-measurement superoperators, and exhibit examples that are causal but not localizable. We construct another class of causal bipartite superoperators that are not localizable by invoking bounds on the strength of correlations among the parts of a quantum system. A bipartite superoperator is said to be semilocalizable if it can be implemented with one-way quantum communication from Alice to Bob, and it is semicausal if it conveys no information from Bob to Alice. We show that all semicausal complete-measurement superoperators are semilocalizable, and we establish a general criterion for semicausality. In the multipartite case, we observe that a measurement superoperator that projects onto the eigenspaces of a stabilizer code is localizable.
The joint expectation values of quantum observables measured by two parties typically depend on the causal structure of the measurement protocol. The two causal structures we investigate here are the direct-cause scenario, in which a single quantum state is measured twice in series with a general quantum channel between measurements, and the common-cause scenario, in which a shared bipartite quantum state is measured in parallel. Through a variety of examples of qubit channels with tunable parameters, we identify the full set of input states where the direct-cause joint expectation values of Pauli observables coincide with those obtained from a common cause, a situation that we call causal confusion. More generally, for arbitrary finite-dimensional quantum systems and light-touch observables, we prove that a channel exhibits causal confusion for some input state if and only if it is a positive-partial-transpose (PPT) channel, thereby endowing PPT channels with a new operational meaning in terms of causal confusion. Using this result, we characterize the set of qubit channels for which there exists an input state leading to causal confusion as the set of entanglement-breaking channels.
A no-signalling channel transforming quantum systems in Alice's and Bob's local laboratories is compatible with two different causal structures: (A⪯BA⪯BA⪯B) Alice's output causally precedes Bob's input and (B⪯AB⪯AB⪯A) Bob's output causally precedes Alice's input. Here I prove that two no-signalling channels that are not perfectly distinguishable in any ordinary quantum circuit can become perfectly distinguishable through the quantum superposition of circuits with different causal structures.
The causal structure of a unitary transformation is the set of relations of possible influence between any input subsystem and any output subsystem. We study whether such causal structure can be understood in terms of compositional structure of the unitary. Given a quantum circuit with no path from input system A to output system B , system A cannot influence system B . Conversely, given a unitary U with a no-influence relation from input A to output B , it follows from [B. Schumacher and M. D. Westmoreland, Quantum Information Processing 4 no. 1, (Feb, 2005)] that there exists a circuit decomposition of U with no path from A to B . However, as we argue, there are unitaries for which there does not exist a circuit decomposition that makes all causal constraints evident simultaneously . To address this, we introduce a new formalism of `extended circuit diagrams', which goes beyond what is expressible with quantum circuits, with the core new feature being the ability to represent direct sum structures in addition to sequential and tensor product composition. A causally faithful extended circuit decomposition, representing a unitary U , is then one for which there is a path from an input A to an output B if and only if there actually is influence from A to B in U . We derive causally faithful extended circuit decompositions for a large class of unitaries, where in each case, the decomposition is implied by the unitary's respective causal structure. We hypothesize that every finite-dimensional unitary transformation has a causally faithful extended circuit decomposition.